I haven't even gotten past page 5 of catching up on this thread..but let's post some of the studies that I have looked at regarding smoking and allergies shall we? Then some people can get off their high horses of false facts and attempt to present actual studies to back up their claims as well:
From
Respiratory Research, 2006, March 7;29
Cigarette smoke exposure facilitates allergic sensitization in mice.Moerloose KB, Robays LJ, Maes T, Brusselle GG, Tournoy KG, Joos GF.
Department of Respiratory Diseases, Ghent University Hospital, Ghent, Belgium.
[email protected]
Abstract:
BACKGROUND: Active and passive smoking are considered as risk factors for asthma development. The mechanisms involved are currently unexplained. OBJECTIVE: The aim of this study was to determine if cigarette smoke exposure could facilitate primary allergic sensitization. METHODS: BALB/c mice were exposed to aerosolized ovalbumin (OVA) combined with air or tobacco smoke (4 exposures/day) daily for three weeks. Serology, lung cytopathology, cytokine profiles in bronchoalveolar lavage fluid (BALF) and on mediastinal lymph node cultures as well as lung function tests were performed after the last exposure. The natural history and the immune memory of allergic sensitization were studied with in vivo recall experiments. RESULTS: Exposure to OVA induced a small increase in OVA-specific serum IgE as compared with exposure to PBS (P < 0.05), while no inflammatory reaction was observed in the airways.
Exposure to cigarette smoke did not induce IgE, but was characterized by a small but significant neutrophilic inflammatory reaction. Combining OVA with cigarette smoke not only induced a significant increase in OVA-specific IgE but also a distinct eosinophil and goblet cell enriched airway inflammation albeit that airway hyperresponsiveness was not evidenced. FACS analysis showed in these mice increases in dendritic cells (DC) and CD4+ T-lymphocytes along with a marked increase in IL-5 measured in the supernatant of lymph node cell cultures. Immune memory experiments evidenced the transient nature of these phenomena. CONCLUSION:
In this study we show that mainstream cigarette smoke temporary disrupts the normal lung homeostatic tolerance to innocuous inhaled allergens, thereby inducing primary allergic sensitization. This is characterized not only by the development of persistent IgE, but also by the emergence of an eosinophil rich pulmonary inflammatory reaction.
Mainstream = the persona actually smoking, not second hand. No proof for secondhand (feel free to look up the entire article, you have all of the info for it)
Would you like me to give you a long drawn out discussion on the relationship between IgE's and allergies? With the VAST knowledge of this that some on here have, I would hope they wouldn't need me to explain
But wait, there's more...many more, let's post another shall we:
Annals of Allergy, Asthma and Immunology, 1999 Feb;82(2):194-7
Abstract:
Tobacco allergy: demonstration of cross-reactivity with other members of Solanaceae family and mugwort pollen.Ortega N, Quiralte J, Blanco C, Castillo R, Alvarez MJ, Carrillo T.
Department of Allergy, Hospital Universitario NTRA SRA del Pino, Las Palmas de Gran Canaria, Canary Islands, Spain.
BACKGROUND:
Tobacco is a plant belonging to the Solanaceae family. This plant is usually used as a contact insecticide for several infestations in some areas, such as the Canary Islands. Allergy induced by inhalation of this plant is unusual. Identification of the potential allergen in growing areas is essential. OBJECTIVE: We report a patient with occupational sensitivity to an aqueous solution of cut tobacco whose clinical manifestations were rhinoconjunctivitis and urticaria. Past medical history was significant for seasonal allergic rhinoconjunctivitis to mugwort pollen and oral allergy syndrome with avocado. METHODS: Green tobacco and cured tobacco leaf extracts were prepared, skin prick tests were performed with green tobacco, cured tobacco leaf extracts, and certain aeroallergens. Conjunctival challenge test was carried out with green tobacco and cured tobacco leaf extract. Serum-specific IgE against tobacco leaf was performed by commercial CAP. CAP inhibition experiments were carried out with tobacco and Artemisia vulgaris. RESULTS: Skin prick tests and conjunctival challenge tests with green tobacco and cured tobacco leaf extracts were positive, as well as serum-specific IgE by CAP, indicating an IgE-mediated sensitization. CAP inhibition experiments were carried out and it was found that tobacco, mugwort pollen, and tomato extracts inhibited the binding of the patient's serum to solid-phase tobacco leaf. No inhibition was observed when Alternaria, D. pteronyssinus, and potato were used as control inhibitors. Inhibition of immunoCAP to mugwort was obtained with mugwort and tobacco extracts and no cross-reactivity to D. pteronyssinus was shown. CONCLUSION: The results suggest that tobacco can induce IgE-mediated reactions that are mediated by the existence of common antigenic epitopes between tobacco and mugwort pollen.
This allergy can be a hazard of employment in the agricultural areas.
That's just two I found on PubMed in about a minute. There are no, and I repeat NO studies that I have ever seen that directly relate second hand smoke and allergies. I agree it's a nasty habit, and it does cause asthma, but that does not change the fact that actual medical studies do not back up the media hype and what people
want to be true.
So before anyone says I have nothing to back up my claim and acts like they are the all knowing one, please present me with any journal articles you have that show a relationship between second hand smoke and allergies (unless you happen to work in the Canary Islands as in the agriculture department, then I'll believe you have an allergy). I would be interested to read the methodology in their study.
Thanks